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Abstract Chronic venous insufficiency is a functional abnormality of the venous system caused by valvular incompetence may or may not be associated with the obstruction of the venous flow. It can affect the superficial venous system, the deep system, or both. In addition venous dysfunction can be the result of a congenital disorder or may be acquired (Porter JM, 1995). The prevalence of chronic venous insufficiency in the population increase with age . On this study the peak incidence of chronic venous insufficiency in lower limbs between 30 -40 years (39.7%), the incidence of males to females was 47.1%-52.9%. Traditionally, chronic venous insufficiency has been grouped into four clinical forms: primary or essential varicose veins, post-thrombotic syndrome, varicose veins due to congenital angioplasties (Kiippel-Trenaunay..) and varicose veins due to acquired arterio- venous fistulas. It is recognized today that varicose veins caused by pregnancy constitute a separate group, with its own etiopathogenic and patho- physiologic characteristics (Luis Henrique G F, 2003). There are two mechanisms for venous hypertension. The first is hydrostatic pressure, related to the hydrostatic pressure of the blood of the right atrium . The second mechanism is dynamic and is related to the calf musculature (muscular pump) that exercises an important role |