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Abstract Polycystic ovary disease (PCOD) is a topic likely to generate greater controversy about etiology or patliogenesis than any other disease in gynecology It remains an enigma because of the superficial nature of our knowledge regarding its etiology and the pathophysiology (Takahashik et al; 1993). Polycystic ovary syndrome is a clinical disorder characterized by infertiliy, oligomenorrhoea, hirsutism, acne or seborrhea and obesity. Recently, it has been suggested that the condition is hereditary and that the women with polycystic ovary syndrome have additional metabolic disturbance further more it can result from disturbance of various endocrine system (YenSSG. 1991). Defining the Polycystic ovarian syndrome has challenged clinicians for many years. The clinical, honnonal and morphological definition of PCOS has their own limitations, and does not correspond exactly (Dewailly D. 1997).Over 10 years after the introduction of pelvic ultrasonography still no agreement that has been reached on criteria used for neither polycystic ovary diagnosis nor its validity (Even J. et al 1997). Regulation of ovarian and uterine blood flow is an important aspect of reproductive physiology Changes in the vasciilature of the cycling ovary are rapid and profound previous studies have suggested that blood flow to individual follicle may play an instrumental role in selection and maturation of the dominant follicle in primates (Zele Znik et al; 1981). |