الفهرس 
Review of LiteratureOnly 14 pages are availabe for public view
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Abstract
The evaluation of thyroid function is commonly conducted to
determine the cause of obesity in children and adolescents. Numerous
studies have shown that obese children have a higher concentration of
the thyroid-stimulating hormone (TSH), compared with their nonobese
peers, TSH correlates positively with body mass index (BMI)
and the degree of obesity expressed by BMI SDS (standard deviation
score).
Thyroid hormones are closely linked to the processes involved in
growth and development and free T4 (FT4) and thyroid-stimulating
hormone (TSH) levels are presumed to be related to obesity. Recently,
there has been an increasing focus on thyroid function in obesity.
Thyroid hormones and TSH concentration are reported to be normal,
elevated, or even low in people with obesity in comparison to normal
weight individuals.
In children with obesity, TSH and thyroid hormone
concentrations are shown to be high, which is accompanied by some
abnormalities in this population, including weight gain, increased
cholesterol levels, impaired growth velocity, anemia, sleepiness,
weakness, and impaired psychomotor and cognitive development.
Although there are various studies evaluating the thyroid hormone and
TSH levels in children with obesity and possible related
complications, the findings are inconsistent.
Hyperthyrotropinemia with normal serum free thyroxine (fT4)
and a normal or slightly elevated level of free triiodothyronine (fT3) is
a common abnormality of thyroid function, more frequently observed
in obese children than in the general population. Subclinical hypothyroidism, resulting from an autoimmune process, is rare in
obese children.