الفهرس 
Introduction and rationaleOnly 14 pages are availabe for public view
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Abstract
This thesis consist of the following parts:
1-Introduction:
This section includes a brief survey about Preeclampsia, ateiology, treatment and of drugs used to treat preeclampsia and the genetic causes of preeclampsia.
2-Research objectives:
This part implicates the rational upon which thisstudy conducted and designed.
3-Clinical examinations:
This part implicates the clinical laboratory examinations which conducted on pregnant in this study including determination of triglycerides, cholesterol, uric acid, ALT, AST,creatinine, urea, CD-36 and endocan.
4-Detection of locus 7q11 rs1761667 polymorphism:
This part describes the practical procedures used forDetection of locus 7q11 rs1761667 polymorphism by restriction fragment length polymorphism (RFLP) method.
5- The Results:
The main results which the research concluded are:
High levels of sCD-36 were associated with preeclampsia, and the minor (A) allele of locus 7q11 genomic region rs1761667 polymorphism was repeated with higher frequency in preeclampsic patients than healthy controls. Patients with AA genotype of rs1761667 were found to have significant increase in blood pressure, proteinuria, triglycerides and total cholesterol. Serum endocan level was also higher in preeclampsia patients than healthy controls. Serum levels of sCD-36 were positively correlated endocan
6- Discussion
This part implicates the possible explanations of the research results and describe how these results are compatible with the previous studies.
5-References:
The thesis includes 155 references covering the period from 1985 to 2019