الفهرس 
Pathophysiology of Septic ShockOnly 14 pages are availabe for public view
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Abstract
Septic Shock is known as tissue hypoperfusion that
occurs due to septicemia from an invading organism leads state
of tissue hypoxia, multi organ failure and death from such
conditions, cleared vision of its Pathophysiology about the
nature of causative organism either bacterial or fungal with or
without viral element, Also about the sequential cascade of the
human immune system that defend against the invading
organism focusing on the immune dysfunction that occur
resulting in progression from septicemia or bacteremia to septic
shock (Kumar et al., 2006).
Septic shock needed to know its main lines of treatment
of EGDT describing the main bulk of the therapy of
intravenous fluids, vasopressors, inotropes, Corticosteroids,
APC and antibiotics beside the supportive treatment control of
anemia and hyperglycemia (Rivers et al., 2001).
Septic shock treatment deals with some conditions that
patients have persistent hypoperfusion despite of the
vasopressor support, here comes the role of corticosteroids in
improving these patients by rising their blood pressure and if
they dependable on mechanical ventilators they can be weaned
preventing their deterioration to ARDS that has high mortality
index compared to other causes of mortality of septic shock.
(Sivabalan et al., 2008)