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Abstract Asherman’s syndrome is defined by the presence of permanent intrauterine adhesions (IUA), formed during the scarring of endometrial surface defects, and obliterating partially or completely the uterine cavity. The presence of such adhesions has been known to be associated with hypomenorrhea or amenorrhea, infertility, and obstetric complications. The most frequent causes of their formation are postpartum or postabortion overzealous dilatation and curettage. Other uterine infections or surgical trauma might also be the predisposing factor (Asherman, 1948, Rabau et al., 1963; Forssman, 1965; Carmichael, 1970;; Schenker et al., 1982; Kurman et al., 1994; and Pabuçcu et al., 1997). Surgical excision of the intrauterine adhesions has evolved over time. In the past, access to the uterine cavity was by direct abdominal approach using laparotomy or via the transcervical route with blind division of adhesions using a uterine sound. This did not allow analyzing results in relation to proper visual assessment of the severity of the adhesions and the extent of uterine cavity occlusion. The use of hysteroscopy in such case since the early 1970s, particular with the newer instrumentation that allows for easier and faster dissection, has allowed rapid improvement in the diagnosis, assessment, and treatment of IUA (March et al.,1978; Sanfilippo et al.,1982; Otubu et al., 1989; Dicker et al., 1996; and Amer et al., 2005). |