الفهرس 
Introduction and Aim of the WorkOnly 14 pages are availabe for public view
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Abstract
Aim of the work
To evaluate the existence of central conduction deficit in diabetic patients and the extent of this dysfunction in relation to degree of the peripheral nerve involvement.
Conclusions
It is concluded that SSEP can suggest an evidence of existence of central nervous system pathology besides the peripheral neuropathy which is documented by nerve conduction study. Also, it is concluded that hyperglycemia represents an important risk factor for both central and peripheral neuropathy, while the duration of diabetes only and not glycemic control was concluded to be very risky for development of central conduction abnormalities. Thus, metabolic factors are accused for peripheral neuropathy rather than central nervous system complications while vascular causes can be suggested as a different underlying cause for central changes.
Recommendations
We recommend follow up of this research with the aid of neuroimaging as well as neuropathology to verify the suggested neuropsychological evidence of central nervous system in diabetes mellitus.